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Vasodilator Action of Guanethidine
Journal article   Open access   Peer reviewed

Vasodilator Action of Guanethidine

Francois Abboud and John Eckstein
Circulation research, Vol.11(5), pp.788-796
11/1962
DOI: 10.1161/01.RES.11.5.788
PMID: 14010558
url
https://doi.org/10.1161/01.RES.11.5.788View
Published (Version of record) Open Access

Abstract

The cardiovascular effects of intravenous guanethidine were measured in animals treated with reserpine. The action of intraarterial guanethidine was studied also in perfused forelegs of dogs. The following conclusions were reached:1. There was no significant increase in cardiac output, heart rate, or systemic arterial pressure following intravenous guanethidine in treated animals; only sustained vasodilatation was observed.2. The vasodilatation was caused by a local action of the drug on vascular smooth muscle. The dilatation became more pronounced following depletion of catecholamines with reserpine and repeated administration of guanethidine.3. The dilatation did not appear to be mediated through the release of catecholamines, histamine, or acetylcholine. It may have resulted, at least in part, from stimulation of beta adrenergic receptors or other “dilator receptors” which are blocked by dichloroisoproterenol.

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