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p16(INK4A) expression in invasive laryngeal cancer
Journal article   Open access   Peer reviewed

p16(INK4A) expression in invasive laryngeal cancer

Brenda Y Hernandez, Mobeen Rahman, Charles F Lynch, Wendy Cozen, Elizabeth R Unger, Martin Steinau, Trevor Thompson, Maria Sibug Saber, Sean F Altekruse, Marc T Goodman, …
Papillomavirus research, Vol.2, pp.52-55
12/2016
DOI: 10.1016/j.pvr.2016.03.001
PMCID: PMC5637278
PMID: 29034342
url
https://doi.org/10.1016/j.pvr.2016.03.001View
Published (Version of record) Open Access

Abstract

We examined p16 expression in tumors from a population-based sample of laryngeal cancer cases diagnosed in the U.S. Samples had been previously genotyped for HPV DNA. Overall, p16 expression was observed in laryngeal tissue from 8 of 101 (7.9%) cases. p16 expression was observed in 2 of 16 (12.5%) cases previously determined to be HPV DNA positive. The two cases dually positive for p16 and HPV DNA were non-keratinizing SCC and papillary SCC tumors that were positive for genotypes 18 and 35/89, respectively. Positivity for p16 and/or HPV DNA was not associated with 5-year survival (log-rank p value=0.55). Our findings support a limited role of HPV in laryngeal carcinogenesis. p16 is not a reliable surrogate for HPV status in laryngeal cancers and is not a predictor of laryngeal cancer survival.
Laryngeal Cancer HPV Human papillomavirus Larynx P16 P16(INK4A)

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