Letter/Communication
Wasp Sting-Associated Occlusion of the Supraclinoid Internal Carotid Artery: Implications Regarding the Pathogenesis of Moyamoya Syndrome
Archives of neurology (Chicago), Vol.46(6), pp.607-608
06/01/1989
DOI: 10.1001/archneur.1989.00520420025018
PMID: 2730372
Abstract
TO THE EDITOR. —The earliest pathologic changes in moyamoya syndrome are stenotic and/or occlusive changes in the terminal portion of the internal carotid artery.1 The cause of moyamoya syndrome, particularly the influence of congenital and/or acquired factors, remains undefined. Suzuki2 has been the chief proponent of a pathogenetic mechanism that involves the interaction of the autonomic nervous system (local factor) and the immune system (systemic factor). We report wasp sting-associated occlusion of the terminal internal carotid artery, and suggest that this may have similar implications regarding the pathogenesis of moyamoya syndrome. REPORT OF A CASE. —A previously healthy, developmentally normal 34-month-old white boy was stung repeatedly on the inner upper lip near the midline. The father removed a live yellow jacket from the child's mouth. There were no stings in the posterior aspect of
Details
- Title: Subtitle
- Wasp Sting-Associated Occlusion of the Supraclinoid Internal Carotid Artery: Implications Regarding the Pathogenesis of Moyamoya Syndrome
- Creators
- Judith T RomanoJack E RiggsJohn B BodensteinerLudwig Gutmann
- Resource Type
- Letter/Communication
- Publication Details
- Archives of neurology (Chicago), Vol.46(6), pp.607-608
- DOI
- 10.1001/archneur.1989.00520420025018
- PMID
- 2730372
- NLM abbreviation
- Arch Neurol
- ISSN
- 0003-9942
- eISSN
- 1538-3687
- Publisher
- American Medical Association
- Language
- English
- Date published
- 06/01/1989
- Academic Unit
- Neurology
- Record Identifier
- 9984020771202771
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