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An injury-induced tissue niche shaped by mesenchymal plasticity coordinates the regenerative and disease response in the lung
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An injury-induced tissue niche shaped by mesenchymal plasticity coordinates the regenerative and disease response in the lung

Dakota L. Jones, Michael P. Morley, Xinyuan Li, Yun Ying, Fabian L. Cardenas-Diaz, Shanru Li, Su Zhou, Sarah E. Schaefer, Ullas V. Chembazhi, Ana Nottingham, …
bioRxiv
Cold Spring Harbor Laboratory, 1.1
02/29/2024
DOI: 10.1101/2024.02.26.582147
PMCID: PMC10962740
PMID: 38529490
url
https://doi.org/10.1101/2024.02.26.582147View
Preprint (Author's original)This preprint has not been evaluated by subject experts through peer review. Preprints may undergo extensive changes and/or become peer-reviewed journal articles. Open Access

Abstract

Severe lung injury causes basal stem cells to migrate and outcompete alveolar stem cells resulting in dysplastic repair and a loss of gas exchange function. This “stem cell collision” is part of a multistep process that is now revealed to generate an injury-induced tissue niche (iTCH) containing Keratin 5+ epithelial cells and plastic Pdgfra+ mesenchymal cells. Temporal and spatial single cell analysis reveals that iTCHs are governed by mesenchymal proliferation and Notch signaling, which suppresses Wnt and Fgf signaling in iTCHs. Conversely, loss of Notch in iTCHs rewires alveolar signaling patterns to promote euplastic regeneration and gas exchange. The signaling patterns of iTCHs can differentially phenotype fibrotic from degenerative human lung diseases, through apposing flows of FGF and WNT signaling. These data reveal the emergence of an injury and disease associated iTCH in the lung and the ability of using iTCH specific signaling patterns to discriminate human lung disease phenotypes.
Cell Biology

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